Indica Labs’ Sales Application Scientist Adam Smith will be presenting on the STRATA Platform July 14th from 11:45-12:15 at the 2nd Digital Pathology Congress in
Join Indica Labs at the 2nd Digital Pathology Congress in Philadelphia Read More »
Indica Labs would like to formally invite you to the Inaugural East Coast HALOTM User Group Meeting which will be held on 11th February, 2016
Inaugural East Coast HALO User Group to be held in Cambridge, MA on February 11, 2016 Read More »
Discoveries from the field: Expediting drug developments and clinical trials with digital pathology and HALO image analysis Alex Klimowicz, PhD, Principal Scientist, Boehringer Ingelheim Pharmaceuticals
File Formats: Non-proprietary (JPG, TIF, OME.TIFF) Nikon (ND2) 3DHistech (MRXS) Akoya (QPTIFF, component TIFF) Olympus / Evident (VSI) Hamamatsu (NDPI, NDPIS) Aperio (SVS, AFI) Zeiss
Supported File Formats for HALO®, HALO AI, and HALO Link Read More »
The TNF-α-induced protein 8 (TNFAIP8 or TIPE) is a risk factor for cancer and bacterial infection, and its expression is upregulated in a number of
Exacerbated Experimental Colitis in TNFAIP8-Deficient Mice Read More »
Infiltration of the tumor microenvironment by nerve fibers is an understudied aspect of breast carcinogenesis. In this study, the presence of nerve fibers was investigated
Recently, it was reported that angiopoietin-like protein 8 (ANGPTL8) was the long-sought “betatrophin” that could control pancreatic beta cell proliferation. However, studies of Angptl8(?/?) mice
ANGPTL8/Betatrophin Does Not Control Pancreatic Beta Cell Expansion Read More »
Islet-1 (Isl-1) is essential for the survival and ensuing differentiation of pancreatic endocrine progenitors. Isl-1 remains expressed in all adult pancreatic endocrine lineages; however, its
Islet-1 is essential for pancreatic beta cell function Read More »
The regulated release of anorexigenic α-melanocyte stimulating hormone (α-MSH) and orexigenic Agouti-related protein (AgRP) from discrete hypothalamic arcuate neurons onto common target sites in the
G-protein-independent coupling of MC4R to Kir7.1 in hypothalamic neurons Read More »
Antagonizing glucagon action represents an attractive therapeutic option for reducing hepatic glucose production in settings of hyperglycemia where glucagon excess plays a key pathophysiological role.
